Presentation Information
Imitators Of Preeclampsia: Manifestations Of Endothelial Dysfunction In Context Of Hypertensive Disorders Of Pregnancy
- Speaker: Jeff M. Denney , MD, MS, FACOG
- Presentation Type:
- Duration: 60 Mins
- Credits: 1 CME
Abstract:
Background: Simply stated, preeclampsia is new-onset hypertension with proteinuria during gestation. However, preeclampsia is complex, virtually affects every organ and is often confused with other entities--imitators of preeclampsia.
Methods: My aim is to provide clinicians with a working framework. Such framework is key in identifying underlying disease processes that mimic or predispose patients toward developing hypertensive disorders and evolving manifestations (eg, renal failure in SLE).
Results: Select data will be reviewed to tie in insights with numerous portals and pathophysiologic processes that lead to pre-eclampsia, hypertensive disorders or pregnancy and the so-called imitators of preeclampsia (eg, AFLP, HELLP, SLE, TTP-HUS). Underlying processes or preexisting chronic diseases all lead to an assortment of physiologic dysfunctions affecting uterus and kidney with or without additional organ involvement.
Conclusion: Many disease processes predispose toward preeclampsia and/or imitators of preeclampsia. Regardless of background and ultimate pathway, over-arching principles are shared.
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15-11-2020 08:00
15-11-2020 09:00
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Imitators Of Preeclampsia: Manifestations Of Endothelial Dysfunction In Context Of Hypertensive Disorders Of Pregnancy
Background: Simply stated, preeclampsia is new-onset hypertension with proteinuria during gestation. However, preeclampsia is complex, virtually affects every organ and is often confused with other entities--imitators of preeclampsia.
Methods: My aim is to provide clinicians with a working framework. Such framework is key in identifying underlying disease processes that mimic or predispose patients toward developing hypertensive disorders and evolving manifestations (eg, renal failure in SLE).
Results: Select data will be reviewed to tie in insights with numerous portals and pathophysiologic processes that lead to pre-eclampsia, hypertensive disorders or pregnancy and the so-called imitators of preeclampsia (eg, AFLP, HELLP, SLE, TTP-HUS). Underlying processes or preexisting chronic diseases all lead to an assortment of physiologic dysfunctions affecting uterus and kidney with or without additional organ involvement.
Conclusion: Many disease processes predispose toward preeclampsia and/or imitators of preeclampsia. Regardless of background and ultimate pathway, over-arching principles are shared.
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